University of California Los Angeles

FRANK S. BARNES* Distinguished Professor Department of Electrical and Computer Engineering University of Colorado BoulderOM P. GANDHI Professor Department of Electrical Engineering at the University of Utah in Salt Lake City – I . Leeka MAILA Hietanen Senior Researcher non-Ionizing Radiation section Finnish Institute of Occupational Health Helsinki Kheifets Professor of Epidemiology School of Public Health, University of California Los Angeles.

DAVID L. MCCORMICK senior vice president and director of the Illinois Institute of Technology Research Institute,the idea ofation Induced Colon Cancer Stopped in his tracks by the TNF-alpha antagonistcolitis people with inflammatory bowel disease colitis are at increased risk of developing colorectal cancer. New data from Naofumi Mukaida and generated generated at Kanazawa University, a central role for the soluble factor, TNF-alpha in the development of colon cancer in mice in which inflammation of the gut was induced by administration of azoxymethane ingestion. Expression of TNF-alpha was in the large intestine of normal mice AOM AOM and DSS increased and this has been the development of tumors in the colon. Where one of the receptors for TNF-alpha and with an antagonist with an antagonist of TNF-alpha protected significantly from the effects of treatment with AOM and DSS development less inflammation of the colon and fewer tumors in the colon. As suggested by the authors , and by Ezra Burstein and Eric R. Fearon in an accompanying commentary, these data clear justification for the idea drugs that antagonize TNF-alpha can be useful in reducing the risk of colon cancer in people with ulcerative colitis.

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